Fig. 1

Inhibition of cancer cell proliferation by cardiac glycosides. HepG2 cells (A) and KB cells (B) were treated with 300 nM ouabain, oleandrin, or digoxin for 24 h. The cell number was counted before (0 h) and after (24 h) treatments of cardiac glycosides. As a control, the cells were cultured in the absence of cardiac glycosides for 24 h (cont). n = 3–6. **P < 0.01 versus control