Fig. 1
From: Ionic mechanisms of ST segment elevation in electrocardiogram during acute myocardial infarction
Heart and torso meshes. a Left: heart meshes with the conduction system in the right anterior oblique (RAO) and left anterior oblique (LAO) views. Right: torso mesh. b Effects of hyperkalemia, acidosis, and adenosine triphosphate (ATP) depletion on sodium (\(I_{\text{Na}}\)), L-type calcium (\(I_{{{\text{Ca}},{\text{L}}}}\)), rapid delayed rectifier potassium (\(I_{\text{Kr}}\)), slow delayed rectifier potassium (\(I_{\text{Ks}}\)), inward rectifier potassium (\(I_{{{\text{K}}1}}\)), ATP-sensitive potassium (\(I_{{{\text{K}}_{\text{ATP}} }}\)), and sodium/potassium ATPase (INaK) currents, and the reversal potential for potassium currents (\(E_{K}\)). Upward arrow: facilitatory effect. Downward arrow: Inhibitory effect. (c) Front (left), top (middle), and short axis (right) views of the heart model. The ischemic condition was applied to the pale-colored region perfused by the anterior descending artery