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Fig. 18 | The Journal of Physiological Sciences

Fig. 18

From: Differential control of efferent sympathetic activity revisited

Fig. 18

Examples of percent changes, relative to pre-stimulation average activities, in chronic recordings of renal sympathetic (RSNA, left) and lumbar sympathetic (LSNA, right) nerve activities together with mean arterial pressure (MAP) in rats on high salt diet as influenced by chronic systemic ANGII infusion (angiotensin II) with preceding control period (control) and subsequent recovery period (recovery). Gradual development of arterial hypertension indicates its neurogenic origin induced by central actions of ANGII at sites where the blood–brain barrier is permeable for the peptide. Courses of renal and lumbar sympathetic activities differ quantitatively and do not explain alone how hypertension is generated. The involvement of enhanced visceral sympathetic activity enhancing venoconstrictor tone is tentatively considered according to hemodynamic evidence provided by [42]. Diagrams taken from Yoshimoto et al. [85]

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